In conservative surgical treatment of PPH, pelvic vessel embolization and compressive sutures are associated not only with high rates of subsequent fertility but also with placental disorders, intrauterine growth restriction, cesarean delivery, and PPH recurrence.
Cognitive behavior therapy not only restored ovarian function in women with functional hypothalamic amenorrhea (FHA) but it also lowered cortisol levels and improved neuroendocrine and metabolic concomitants of FHA.
Small glutamine-rich tetratricopeptide repeat–containing protein alpha is expressed in human ovarian tissues and has the potential to modulate androgen receptor signaling, but expression levels may not be abnormal in polycystic ovary syndrome.
Rate of ovarian primordial follicle recruitment increases prepubertally then declines again following the onset of puberty, while ovarian follicle activity continues to increase. These changes seem to track with the age of menarche.
Ganglionic cholinergic stimulation produces changes in ovarian release of progesterone, gonadotropin-releasing hormone, noradrenaline, and adrenaline; the ovarian nerve plexus regulates these mechanisms through the release of different neurotransmitters.
Among 62 women with diminished ovarian reserve, self-esteem significantly predicted fertility distress both directly and indirectly through emotional reactions to potentially being a fragile X premutation carrier.
Gonadotropin-releasing hormone agonist and/or an aromatase inhibitor treatment stabilized or improved disease in patients with benign metastasizing leiomyoma; a selective progesterone receptor modulator was associated with improved symptoms despite increasing fibroid size.
Exploratory genetic analysis of functional polymorphisms 5-HTTLPR and rs6295 reveals clinical implications during early pregnancy loss events in recipients undergoing IVF treatments using donated oocytes.
Natural killer and T-regulatory cells participate in the pathogenesis of endometriosis; establishing the role of certain chemokines in modulating this response represents a fundamental key to understanding the inflammatory response.
Mitochondrial displacement (D-) loop alterations may constitute an inheritable risk factor for endometriosis. The analysis of D-loop alterations might help to identify patients at high risk for disease outcome.
Multidrug resistance–associated protein 4 expression is augmented in peritoneal endometriotic lesions, where it localizes to glandular epithelial cells and is inhibited by lipoxin A4 in endometriotic epithelial cells in vitro.
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