In women with endometriosis and pain in the anterior-lateral part of the thigh, neurologic examination and skin biopsy could be an important way to diagnose nerve invasion, guiding suitable treatments.
Vanishing twins demonstrate significantly lower early b-hCG level increases than normal singleton or twin pregnancies; increases, however, are within clinically normal limits. Abnormal early b-hCG level increases should not be attributed to a vanishing twin.
Impaired prolactin (PRL) expression in samples from miscarriages and the particular expression of the proinflammatory cytokines suggest the pivotal role of PRL in vital pregnancies, reciprocally affected by interleukin-2 expression.
The Blastocyst Euploid Selective Transfer (BEST) Trial demonstrated that single euploid blastocyst transfer results in ongoing pregnancy rates that are equivalent to transferring two untested blastocysts, while dramatically reducing the risk of multiple gestations.
Investigation of 39 men with idiopathic infertility revealed a heterozygous partial deletion of the CLCA4 gene in one patient with cryptozoospermia, making CLCA4 a novel candidate gene for male factor infertility.
Similar fertilization and pregnancy rates were found in ICSI cycles involving men with markedly high DNA damage (DNA fragmentation index >50) compared with healthy men and controlled for partner variables.
We assessed sperm DNA fragmentation (TUNEL) at different time periods before ICSI, and found DNA fragmentation significantly decreased after gradient and samples with TUNEL >20% were more susceptible to a significant increase over time.
This prospective study suggests that intracytoplasmicmorphologically selected sperm injection is effective in patients with severe teratozoospermia but unsuccessful in patients with repeated intracytoplasmic sperm injection failure in the absence of severe male factor.
Ovarian hyperandrogenism, hyperinsulinemia from insulin resistance, and altered intrafollicular paracrine signaling influence ovarian morphology, follicle survival, and growth as well as oocyte development in women with polycystic ovary syndrome.
Potential implications of alterations of the Kiss1 system in the pathophysiology of polycystic ovary syndrome are discussed. The limited experimental and clinical evidence supporting such a role is summarized.
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